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Dr. Stephen Welty

Stephen E. Welty, M.D.

Academic Appointments

  • Professor of Pediatrics—Neonatology
  • Head, Section of Neonatology

Clinical Titles

  • Chief, Neonatology Service, Texas Children's Hospital

Board Certifications

  • Pediatrics
  • Neonatal–Perinatal Medicine

Current Positions in Professional Organizations

  • Member, American Academy of Pediatrics
  • Member, Society for Pediatric Research
  • Member, Perinatal Research Society
  • Member, American Thoracic Society
  • Member, AAAS

Professional History

  • M.D., Oregon Health Sciences University, Portland, OR, 1982.
  • Pediatric Resident, Baylor College of Medicine, Houston, TX, 1983–1985.
  • Post-doctoral Fellow in Neonatal-Perinatal Medicine, Baylor College of Medicine, Houston, TX, 1985–1986, 1987-1990.

Academic Interests

  • Prevention and management of chronic lung disease in preterm infants
  • Gene expression and regulation in neonatal oxidant stress states
  • Mechanisms and significance of neonatal lung inflammatory diseases

Recent Publications

  1. Velten M, Heyob KM, Rogers LK, Welty SE. Deficits in lung alveolarization and function after systemic maternal inflammation and neonatal hyperoxia exposure. J Appl Physiol. 2010 108:1347-56.
  2. Rogers LK, Tipple TE, Britt RD, Welty SE. Hyperoxia exposure alters hepatic eicosanoid metabolism in newborn mice. Pediatr Res. 2010 67:144-149.
  3. Tipple TE, Welty SE, Nelin LD, Hansen JM, Rogers LK. Alterations of the thioredoxin system by hyperoxia: implications for alveolar development Am J Respir Cell Mol Biol. 2009 41:612-619.
  4. Rogers LK, Tipple TE, Nelin LD, Welty SE. Differential responses in the lungs of newborn mouse pups exposed to 85% or >95% oxygen. Pediatr Res. 2009 65:33-38.
  5. Heyob KM, Rogesr LK and Welty SE. Transgenic Mice with Glutathione Reductase Targeted to the Mitochondria of Type II Cells Are Not Riboflavin Deficient in Hyperoxia, Am J Respir Crit Care Med 179;2009:A5004.