Stephen E. Welty, M.D.
- Professor of Pediatrics—Neonatology
- Head, Section of Neonatology
- Chief, Neonatology Service, Texas Children's Hospital
- Neonatal–Perinatal Medicine
Current Positions in Professional Organizations
- Member, American Academy of Pediatrics
- Member, Society for Pediatric Research
- Member, Perinatal Research Society
- Member, American Thoracic Society
- Member, AAAS
- M.D., Oregon Health Sciences University, Portland, OR, 1982.
- Pediatric Resident, Baylor College of Medicine, Houston, TX, 1983–1985.
- Post-doctoral Fellow in Neonatal-Perinatal Medicine, Baylor College of Medicine, Houston, TX, 1985–1986, 1987-1990.
- Prevention and management of chronic lung disease in preterm infants
- Gene expression and regulation in neonatal oxidant stress states
- Mechanisms and significance of neonatal lung inflammatory diseases
- Velten M, Heyob KM, Rogers LK, Welty SE. Deficits in lung alveolarization and function after systemic maternal inflammation and neonatal hyperoxia exposure. J Appl Physiol. 2010 108:1347-56.
- Rogers LK, Tipple TE, Britt RD, Welty SE. Hyperoxia exposure alters hepatic eicosanoid metabolism in newborn mice. Pediatr Res. 2010 67:144-149.
- Tipple TE, Welty SE, Nelin LD, Hansen JM, Rogers LK. Alterations of the thioredoxin system by hyperoxia: implications for alveolar development Am J Respir Cell Mol Biol. 2009 41:612-619.
- Rogers LK, Tipple TE, Nelin LD, Welty SE. Differential responses in the lungs of newborn mouse pups exposed to 85% or >95% oxygen. Pediatr Res. 2009 65:33-38.
- Heyob KM, Rogesr LK and Welty SE. Transgenic Mice with Glutathione Reductase Targeted to the Mitochondria of Type II Cells Are Not Riboflavin Deficient in Hyperoxia, Am J Respir Crit Care Med 179;2009:A5004.